- Challenges
- (I) Multiple sclerosis: Genuine naked-eye identification
- (I-1) Pioneering spinal cord findings
- (I-2) Detection of distinctive brain lesions
- (I-3) Macropathology: Unexploited key evidence
- (II) Histological perspectives
- (II-1) Tissue changes in specific lesions
- (II-2) Histological lesion categorizations
- (II-3) “Selective demyelination”: The facts
- (II-3-a) Primary demyelinations: A commonplace phenomenon
- (II-3-b) Reverberations of a histological misspecification
- (II-3-c) The root of all evil: Demyelination
- (II-3-d) Lymphocytic infiltrations: Active, or re-active?
- Immunocytes and “myelophages”: Rare, or late to appear
- Autoimmunity: Not solving problems, only raising questions
- (II-3-e) Primary inflammation: Adding to the confusion
- (II-3-f) Histology: Not the key to understanding multiple sclerosis
- (III) The neurologist’s standpoint
- (III-1) Classical clinical observations
- (III-2) CDMS: Chronically delusive misidentification syndrome
- (IV) The specter of the multiple sclerosis agent
- (IV-1) Lawless or specific pattern(s) of spread?
- (IV-2) From randomly spread foci to multiple sclerosis-agent
- (IV-3) Looking back in frustration
- (V) Lesion explanation in physical terms
- (V-1) The dynamics of lesion formation
- (V-2) Cause of the injurious impulses
- (V-3) The key to decoding multiple sclerosis: Specific data
- References
- Overview of Plates
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